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In medicine (gastroenterology), ascites (also known as peritoneal cavity fluid, peritoneal fluid excess, hydroperitoneum or more archaically as abdominal dropsy) is an accumulation of fluid in the peritoneal cavity. Although most commonly due to cirrhosis and severe liver disease, its presence can portend other significant medical problems.
Diagnosis of the cause is usually with blood tests, an ultrasound scan of the abdomen and direct removal of the fluid by needle or paracentesis (which may also be therapeutic). Treatment may be with medication (diuretics), paracentesis or other treatments directed at the cause.





Signs and symptoms

Mild ascites is hard to notice, but severe ascites leads to abdominal distension. Patients with ascites generally will complain of progressive abdominal heaviness and pressure as well as shortness of breath due to mechanical impingement on the diaphragm.
Ascites is detected on physical examination of the abdomen by visible bulging of the flanks in the reclining patient ("flank bulging"), " shifting dullness " (difference in percussion note in the flanks that shifts when the patient is turned on the side) or in massive ascites with a "fluid thrill" or "fluid wave " (tapping or pushing on one side will generate a wave-like effect through the fluid that can be felt in the opposite side of the abdomen).
Other signs of ascites may be present due to its underlying etiology. For instance, in portal hypertension (perhaps due to cirrhosis or fibrosis of the liver) patients may also complain of leg swelling, bruising, gynecomastia, hematemesis, or mental changes due to encephalopathy. Those with ascites due to cancer (peritoneal carcinomatosis) may complain of chronic fatigue or weight loss. Those with ascites due to heart failure may also complain of shortness of breath as well as wheezing and exercise intolerance.

Classification

Ascites exists in three grades:

  • Grade 1: mild, only visible on ultrasound
  • Grade 2: detectable with flank bulging and shifting dullness
  • Grade 3: directly visible, confirmed with fluid thrill

Remedies & Symptoms


ANTHRACINUM

  • Inturation of cellular tissue.
  • Red lines, streaks and stripes mark out the course of the lymphatics.
  • Oedema of the affected parts.
  • Discharge of ichorous, foul- smelling pus.
  • Gangrene, absorption of ichor with ichoraemia collapse.
  • Violent burning pains, not relieved by Arsenic.
HEPAR SULPER

  • If given early will sometimes about anthrax, later it promotes suppuration.
  • Carbuncle surrounded by anthrax, later it promotes suppuration.
  • Carbuncle surrounded by indurated spots, pain intense, sleeplessness.
  • Stinging burning of edges of ulcer with corroding discharge.

RHUS TOX

  • The parts shaves a blush gangrenous appearance.
  • Itching, burning around the carbuncle, with vertigo, stupor, pale face.
  • Great restlessness; patient feels better while moving about.
  • More indicated in the beginning, when the pains are intense and the affected parts are dark red.

TARENTULA CUBANA

  • Anthrax,, especially on back of neck, with burning excruciating pain.
  • Eeysipelatous redness around the carbuncle.
  • Rigors followed by burning fever, great thirst anxiety, headache, delirium.
  • Profuse perspiration and retention of urine, intense pains in carbuncle.

Anthrax is an acute disease in humans and animals caused by Bacillus anthracis, which is highly lethal in all forms. There are effective vaccines against anthrax, and some forms of the disease respond well to antibiotic treatment.

The anthrax bacillus is one of only a few that can form long-lived spores: in a hostile environment, caused perhaps by the death of an infected host or extremes of temperature, the bacteria produce inactive dormant spores which can remain viable for many decades and perhaps centuries. These spores are found on all continents except Antarctica. When spores are inhaled, ingested, or come into contact with a skin lesion on a host they reactivate and multiply rapidly.


Anthrax most commonly infects wild and domesticated herbivorous mammals which ingest or inhale the spores while eating grass or browsing. Ingestion is assumed to be the most common route by which herbivores contract anthrax, but this is yet to be proven. Carnivores living in the same environment may ingest infected animals and become infected themselves. Anthrax can also infect human when they are exposed to blood and other tissues from infected animals (via inhalation or direct inoculation through broken skin), eat tissue from infected animals, or are exposed to a high density of anthrax spores from an animal's fur, hide, or wool.

Remedies & Symptoms

ANTHRACINUM:

  • Induration of cellular tissue.
  • Red lines, streaks and stripes mark out the course of the lymphatics.
  • Oedema of the affected parts.
  • Discharge of ichorous, foul-smelling pus.
  • Gangrene, absorption of ichor with ichoraemia, collapse.
  • Violent burning pains, not relived by Arsenic.

APIS MILL:

  • Stitching, burning pain in anthrax, with sensitiveness to the touch and erysipelatous redness around it.
  • Furuncles, with manifold sloughs of dead connective tissues.

ARSENIC ALB:

  • Anthrax burning like fire.
  • Intense burning in the seat of the carbuncle and some distance around the tumour.
  • Sensation as if boiling water was running beneath the skin.
  • Restlessness, thirst, debility; at night, from warmth.

HEPAR SULPH.

  • If given early will sometimes about anthrax, later it promotes suppuration.
  • Carbuncle surrounded by indurated sports, pain intense, sleeplessness.
  • Stinging burning of edges of ulcer with corroding discharge.

LACHESIS

  • Dark redness around the sore, which discharges dark, bloody pus.
  • Tension of the skin around the carduncle, as if too short; nightly burning of the ulcer, obliging one to rise and wash it with cold water.
  • Gangrene, carbuncle from blood- poisoning.

Anemia is also known as anæmia/anaemia is defined as a qualitative deficiency of hemoglobin or quantitative deficiency of hemoglobin, a protein found inside red blood cells (RBCs). Since hemoglobin normally carries oxygen from the lungs to the tissues, anemia leads to hypoxia (lack of oxygen) in organs. Since all human cells depend on oxygen for survival, varying degrees of anemia can have a wide range of clinical consequences. Anemia is also caused by a lack of iron in the body.



The three main classes of anemia include excessive blood loss, acutely such as a hemorrhage or chronically through low-volume loss, excessive blood cell destruction Hemolysis or deficient red blood cell production (ineffective hematopoiesis ).
Anemia is the most common disorder of the blood. There are several kinds of anemia, produced by a variety of underlying causes. Anemia can be classified in a variety of ways, based on the morphology of RBCs, underlying etiologic mechanisms, and discernible clinical spectra.

There are two major approaches: The "kinetic" approach which involves evaluating production, destruction and loss, and the "morphologic" approach which groups anemia by red blood cell size. The morphologic approach uses a quickly available and cheap lab test as its starting point (the MCV ).

Signs of Anemia

Anemia goes undetected in many people, and symptoms can be small and vague. Most commonly, people with anemia report a feeling of weakness or fatigue in general or during exercise, general malaise and sometimes poor concentration. People with more severe anemia often report dyspnea which means shortness of breath, on exertion. Very severe anemia prompts the body to compensate by increasing cardiac output, leading to palpitations and sweatiness, and to heart failure.

Pallor (pale skin, mucosal linings and nail beds) is often a useful diagnostic sign in moderate or severe anemia, but it is not always apparent. Other useful signs are cheilosis and koilinychia .

Pica, means the consumption of non-food based items such as dirt, paper, wax, grass, ice, and hair, may be a symptom of iron deficiency, although it occurs often in those who have normal levels of hemoglobin.
Chronic anemia may result in behavioral disturbances in children as a direct result of impaired neurological development in infants, and reduced scholastic performance in children of school age.


Remedies & Symptoms

ALUMINA

  • Anaeima and chlorosis in young girls at puberty.
  • After menses, exhausted physically and mentally, scarcely able to speak .
  • Abnormal carving for indigestible things.
  • Profuse albuminous leucorrhoea.

ARSENIC ALB.

  • Anaemia due to disintegration of the blood-corpuscles.
  • Rapid excessive prostration, with sinking of the vital forces; oedema.
  • Emaciation, wants to be a warm room.
  • Debility from overtaxing muscular tissue by prolonged exertion.
  • Extreme restlessness and fear of death (pernicious anaemia).

HELONIAS

  • Anemia and atony from prolonged haemorrhage, esp. from uterine atony.
  • Debility and languor from affection of the genitor-urinary.
  • Atony of all the organs from indolence and luxury.

NATRUM MUR:

  • Anaemia from loss of fluid, malarious cashexia blood impoverished.
  • Skin harsh, dry yellow.
  • Great exhaustion from any little exertion of mind or body.
  • Palpitation, with sensation as if a bird’s wing were fluttering in left chest.
  • Great emaciation, losing flesh while living well.

BIOCHEMIC REMEDIES:

CALC PHOS:

  • This remedy act by supplying new blood cells.
  • Pains and cramps dependent on anaemic condition, waxy appearance of skin, chlorosis, headache and ringing in ears.
  • Also useful in pernicious anaemic.

SILICEA:

  • Anaemia in infants, when improperly nourished, thin, delicate and puny, inter- currently with other remedies indicated.

Cholera, is also known as Asiatic or epidemic choler. It is an infectious gastroenteritis caused by enterotoxin -producing strains of the bacterium Vibrio cholerae Transmission to humans occurs through eating food or drinking water contaminated with cholera vibrios. The major reservoir for cholera was long assumed to be humans themselves, but considerable evidence exist that aquatic environment can serve as reservoirs of the bacteria.



In its most severe forms, cholera is one of the most rapidly fatal illnesses known, and a healthy person's blood pressure may drop to hypotensive levels within an hour of the onset of symptoms; infected patients may die within three hours if medical treatment is not provided. In a common scenario, the disease progresses from the first liquid stool to shock in 4 to 12 hours, with death following in 18 hours to several days, unless oral rehydration therapy is provided.

Remedies & Symptoms

Symptoms of Cholera include nosebleed, rapid pulse, dry skin, tiredness, abdominal, cramps, leg cramps, nausea and vomiting.
The principal symptom of infection is diarrhea, which is watery and brown at first, but quickly changes to large volumes of pale fluid stools ('rice-water stools'). In the most severe cases dramatic fluid loss from the continuous diarrhea can lead to hypovolemic shock and collapse within 1 to 4 hours. Depending upon the treatment provided, unconsciousness and death can occur anytime from 12 to 18 hours afterwards, although some individual cases may persist for several days.


ACONOITE

  • Caused by fear, fright, shock, heat of the sun, sudden checked of sweat etc.
  • Sudden and violent.
  • Stool rice-watery with vomiting.
  • Bluish face, with black lips, anguish or imbecility in the face, cold limbs, with blue nails.
  • Collapse out of proportion to the evacuations, great and sudden sinking of strength, but with no alarm.
  • Thirst, burning for large quantities of cold water.

CARBO VEG

  • From spoiled fish or meats, fats, salts meats, or getting overhead, etc.
  • Type cholera haemorrahagica, cholera asiatica, where the RBC passes out with serums and tinge is red.
  • Stool- rice-watery, offensive, passes frequently and involuntarily, associated with flatulence.
  • Diarrhoea and vomiting, with oppression of chest and coma, or algid state, collapse without stool.
  • Nose, cheeks and finger- tips bluish, cold tongue, desire to be constantly fanned.

VERATRUM ALBUM

  • For the cases with very profuse, evacuation, profuse vomiting and purging and profuse cold sweat.
  • Server cutting pains in abdomen.
  • Violent diarrhea, with greenish, watery, flocculent stools, followed by rapid prostration.
  • Cramps in calves.
  • Small, almost imperceptible pulse.
  • Hoarse weak, weak, voices, cold breathe.
  • Cold sweat over whole body .

The stye is also known as Hordeolum it is an infection of the sebaceous glands at the base of the eyelashes. These are similar to Chalazia but tend to be of smaller size and feel much more painful and usually produce no lasting damage.



Cause

The caused of Styes are generally by a staphylococcus aureus bacterial infection. Although they are particularly common in infants, styes are experienced by people of all ages. Styes can be triggered by stress or poor nutrition. Using the same razor to shave hair near both the eyes and a mustache can also spread staphylococcus bacteria, potentially leading to styes or other eye infections. The bacteria are contagious, so care should be taken to avoid touching the eye or sharing cosmetics, towels, or washcloths. Styes will last up to 3 weeks without treatment, with treatment up to a week.




Remedies & Symptoms

The first signs of a stye are tenderness, pain, and redness in the affected area. Later symptoms include itching, swelling, watering of the eye, sensitivity to light, and discomfort when blinking. A yellowish bump sometimes develops in the affected area. This can be found on the top, bottom, or any area pertaining to the eye.

HEPAR SULPH

  • Where there is a tendency to a recurrence of the complaint, suppuration inevitable.
  • Unhealthy skin , every little injury inclines to suppurate.

MERCURIUS

  • Tumors hard, inflamed and painful .
  • The abscess matures very slowly.
  • Profuse sweating without relief.

STAPHYSAGRIA

  • Frequently appearing styes.
  • Margin of lids dry with hardness styes or tarsal tumours, sometimes ulcerating.

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